I wish I was a dab hand at grafix, however I'll try to
paint a picture with words.
The old antipsychotics worked by blocking dopamine
receptors, the new ones do the same but also do some
stuff to the seretonin sites too.
First off lets talk about a neurone simplified it consists
of a big roundish blob called the cell body and a long stalk
called an axon. On the cell bodies surface are tree like
structures called dendrites these are in addition to the axon.
Now, lets talk about 2 of these neurons (nerve cells)
the axon from one nerve cell ALMOST connects
to the dendrites of the next nerve cell. I say almost
because theres a tiny gap called a synapse.
The synapse is where the chemical messengers
such as dopomine pass a signal from the end of
the axon to the tip(s) of a dendrite.
Now different synapses around the brain have
different chemical messengers, this is because
the brain is awash with all sorts of hormones and
"neurotransmitters"(chemical messangers) and
the dendrite needs to know that the message it gets
comes from the right place.
When the second cell gets enough dopamine
it "fires" sends an electical impulse down the
axon which makes it release neurotransmitters
down to the next cell on the network.
Now lets take a look at the dendrites.
If you taks APs (anti Psychotics) they are chemicals that fit into
the receptors on the dendrites and stop it
receiving. This effectively shuts down all
downstream communications on the network.
However the brain is a clever old thing
and says hang on a sec ~I havent heard
from charlie lately, so a tiny reverse
synaptic transfer occurs from cell 3
in this sequence that tells cell 2
hey mat cant you grow some more dendrites
you seem to be getting a bit deaf.
Cell 2 says oh yeah, I've been oversleeping
a bit wheres my mate cell 1 he usaully has a chat
hasent been in touch for ages.
So cell 2 grows more dendrites to make
up for the fact that most of them are clogged
up with AP shit.
So you need more and more APs
to clog up more and more receptors.
THats why you shouldn't give APs to
normaloes cause it'll send them mad,
by cuasing them to become hypersensitive
to dopamine.
It's also why theres no cure cause
the treatment makes you worse in
the long run.
But we're schizos, so it dosent matter fuck,
if we get madder 'cause they can just up
thier AP crap as often as they like until
they shoot your liver, or your brain
gives out completely and you get
tardith dementia.
By which time your mum and dad are
dead and they can get one more
fuckhead in a body bag and
stop them sucking off the state.
On their way home they laugh
away in thier mercedes
"Fooled them suckers,
silly buggers don't realise
I'm cuasing all the symptoms.
My, this is the gavy train,
I think I'll put this weeks xs
in elly lilly."
--
WARNING!
When trying to withdraw from many psychiatric drugs, patients can develop serious and even
life-threatening emotional and physical reactions. In short, it is dangerous not only to
start taking psychiatric drugs but also can be hazardous to stop taking them. Therefore,
withdrawal from psychiatric drugs should be done under clinical supervision. Principles of
drug withdrawal are discussed in Your Drug May Be Your Problem: How and Why to Stop Taking
Psychiatric Medications.
information on Prozac and Prozac-like drugs can be found in Talking Back to Prozac by
Peter R. Breggin, M.D. and Ginger Ross Breggin.
DSM IV is the fabrication upon which
psychiatry seeks acceptance by medicine
in general. Insiders know it is more
a political than scientific document.
To its credit it says so
--Loren R. Mosher, M.D.